Clinical Physiology of Circulation

Chief Editor

Leo A. Bockeria, MD, PhD, DSc, Professor, Academician of Russian Academy of Sciences, President of Bakoulev National Medical Research Center for Cardiovascular Surgery


The diagnostics of cerebral venous ischemia

Authors: S.E. Semenov 1 , M.V. Shumilina 2 , E.A. Zhuchkova 1 , A.S. Semenov 3

Company:
1 Research Institute for Complex Issues of Cardiovascular Diseases, Sosnovyy bul’var, 6, Kemerovo, 650002, Russian Federation;
2 A.N. Bakoulev Scientific Center for Cardiovascular Surgery, Rublevskoe shosse, 135, Moscow, 121552, Russian Federation;
3 Practice Wolfgang Theobald Specialist for Radiology, ul. Lothringer, 31, Saarlouis, 66740, Germany

E-mail: Сведения доступны для зарегистрированных пользователей.

Link: Clinical Physiology of Blood Circulaiton. 2015; (): -

Quote as: Semenov S.E., Shumilina M.V., Zhuchkova E.A., Semenov A.S.. The diagnostics of cerebral venous ischemia. Klinicheskaya Fiziologiya Krovoobrashcheniya (Clinical Physiology of Circulation, Russian journal). 2015; 2: 5-16 (in Russ.)

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Abstract

Venous congestion is the primary factor affecting the brain with cerebral venous sinustrombosis is found at cardioembolic stroke in the perifocal ischemic focus area to the damage of the venous outflow, leading to vasogenic edema early development. Ischemia thus develops at the expense of extravasal compression of the supply region of stasis vessels, and by increasing peripheral resistance and decrease in perfusion pressure and is, in fact, a "venous". Transient ischemia in migraine is also associated with impaired venous drainage and stagnant plethora. Periodic outflow in straight sinus and phlebohypertension in cases of right-left cardiac shunt at atrial septal defects and abnormalities of cerebral venous collectors in majority of patients with migraine suggests that venous congestion plays not last role in the pathogenesis of migraine. Foci detected in venous ischemia in the brain during tomographic imaging techniques are often reversible, unlike purely arterial origin. The distinctive clinical manifestation of these states is a headache, despite the variety, and certainly features of venous stasis. Such simple assessment tools such as visual analogue scale headaches and ultrasound parameters arteriovenous ratio showed the greatest predictive value for the diagnosis of venous ischemia.


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